Tired

Answer the Following Questions for Parts IàIV.

Part I:

1. What vital signs or symptoms does Annie exhibit?

2. Can you see any common features in Annie's signs and symptoms?

3. Why is Annie having problems breathing?

4. What are the possible reasons for Annie's condition?

Part II:

1. What additional vital signs or symptoms does Annie exhibit?

2. From the internet, find a picture that shows the mammalian spinal cord in cross section. Include the url where you found it and a statement of why you feel this is a good example.

3. How could a decline in muscle function contribute to eye strain and blurred vision?

4. What are the possible reasons for Annie's condition?

Part III:

1. According to the conduction test, was Annie's nerve function normal?

2. According to Annie's EMG test, was Annie's skeletal muscle function normal?

3. Considering your answers to Questions 1 and 2, why did activity in Annie's motor nerves produce a skeletal muscle response that fatigued during repetitive stimulation?

4. Create a table that replicates Figure 1 and fill it out completely.

5. Now take each stage in turn and discuss how sustained neural activity could create a dysfunction and result in a decrease in muscle response during repetitive motor nerve stimulation.

Part IV:

1. What is the role of the thymus in the body?

2. What is an antigen?

3. Go back to Figure 1, look at each stage, and predict where the antibodies in Annie's blood could act to decrease synaptic function at the neuromuscular junction.

4. Neostigmine is one drug that may be prescribed for people with myasthenia gravis. This drug, like the edrophonium chloride injection, will make Annie feel stronger. Go back to the flow diagram, look at each stage, and determine how these drugs could work to increase synaptic performance.

5. In the absence of treatment, what has happened to the amplitude of Annie's endplate...